1. Nomura, D. K., Long, J. Z., Niessen, S., Hoover, H. S., Ng, S.-W., & Cravatt, B. F. (2010). Monoacylglycerol lipase regulates a fatty acid network that promotes cancer pathogenesis. Cell, 140, 49–61.

2. Kuhajda, F. P. (2000). Fatty-acid synthase and human cancer: New perspectives on its role in tumor biology. Nutrition, 16, 202–208.

3. Guzman, M. (2010). A new age for MAGL. Chemistry & Biology, 17, 4–6.

4. Santos, C. R., & Schulze, A. (2012). Lipid metabolism in cancer. FEBS Journal, 279, 2610–2623.

5. Medes, G., Thomas, A., & Weinhouse, S. (1953). Metabolism of neoplastic tissue. IV. A study of lipid synthesis in neoplastic tissue slices in vitro. Cancer Research, 13, 27–29.

6. Szutowicz, A., Kwiatkowski, J., & Angielski, S. (1979). Lipogenetic and glycolytic enzyme activities in carcinoma and nonmalignant diseases of the human breast. British Journal of Cancer, 39, 681–687.

7. Swinnen, J. V., Brusselmans, K., & Verhoeven, G. (2006). Increased lipogenesis in cancer cells: New players, novel targets. Current Opinion in Clinical Nutrition and Metabolic Care, 9, 358–365.

8. Milgraum, L. Z., Witters, L. A., Pasternack, G. R., & Kuhajda, F. P. (1997). Enzymes of the fatty acid synthesis pathway are highly expressed in in situ breast carcinoma. Clinical Cancer Research, 3, 2115–2120.

9. Menendez, J. A., & Lupu, R. (2007). Fatty acid synthase and the lipogenic phenotype in cancer pathogenesis. Nature Reviews Cancer, 7, 763–777.

10. Swinnen, J. V., Heemers, H., Deboel, L., Foufelle, F., Heyns, W., & Verhoeven, G. (2000). Stimulation of tumor-associated fatty acid synthase expression by growth factor activation of the sterol regulatory element-binding protein pathway. Oncogene, 19, 5173–5181.

11. Van de Sande, T., De Schrijver, E., Heyns, W., Verhoeven, G., & Swinnen, J. V. (2002). Role of the phosphatidylinositol 3′-kinase/PTEN/Akt kinase pathway in the overexpression of fatty acid synthase in LNCaP prostate cancer cells. Cancer Research, 62, 642–646.

12. Wang, H. Q., Altomare, D. A., Skele, K. L., Poulikakos, P. I., Kuhajda, F. P., Di Cristofano, A., et al. (2005). Positive feedback regulation between AKT activation and fatty acid synthase expression in ovarian carcinoma cells. Oncogene, 24, 3574–3582.

13. Menendez, J. A., Mehmi, I., Verma, V. A., Teng, P. K., & Lupu, R. (2004). Pharmacological inhibition of fatty acid synthase (FAS): A novel therapeutic approach for breast cancer chemoprevention through its ability to suppress Her-2/neu (erbB-2) oncogene-induced malignant transformation. Molecular Carcinogenesis, 41, 164–178.

14. Priolo, C., Tang, D., Brahamandan, M., Benassi, B., Sicinska, E., Ogino, S., et al. (2006). The isopeptidase USP2a protects human prostate cancer from apoptosis. Cancer Research, 66, 8625–8632.

15. Yecies, J. L., & Manning, B. D. (2010). Chewing the fat on tumor cell metabolism. Cell, 140, 28–30.

16. Cravatt, B. F., Giang, D. K., Mayfield, S. P., Boger, D. L., Lerner, R. A., & Gilula, N. B. (1996). Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides. Nature, 384, 83–87.

17. Matsuda, L. A., Lolait, S. J., Brownstein, M. J., Young, A. C., & Bonner, T. I. (1990). Structure of a cannabinoid receptor and functional expression of the cloned cDNA. Nature, 346, 561–564.

18. Munro, S., Thomas, K. L., & Abu-Shaar, M. (1993). Molecular characterization of a peripheral receptor for cannabinoids. Nature, 365, 61–65.

19. Pertwee, R. G., Howlett, A. C., Abood, M. E., Alexander, S. P., Di Marzo, V., Elphick, M. R., et al. (2010). International union of basic and clinical pharmacology. LXXIX. Cannabinoid receptors and their ligands: Beyond CB(1) and CB(2). Pharmacological Reviews, 62, 588–631.

20. Mackie, K. (2006). Cannabinoid receptors as therapeutic targets. Annual Review of Pharmacology and Toxicology, 46, 101–122.

21. Hermanson, D. J., & Marnett, L. J. (2011). Cannabinoids, endocannabinoids, and cancer. Cancer and Metastasis Reviews, 30, 599–612.

22. Gomez del Pulgar, T., Velasco, G., Sanchez, C., Haro, A., & Guzman, M. (2002). De novo-synthesized ceramide is involved in cannabinoid-induced apoptosis. Biochemical Journal, 363, 183–188.

23. Blazquez, C., Salazar, M., Carracedo, A., Lorente, M., Egia, A., Gonzalez-Feria, L., et al. (2008). Cannabinoids inhibit glioma cell invasion by down-regulating matrix metalloproteinase-2 expression. Cancer Research, 68, 1945–1952.

24. Long, J. Z., Li, W., Booker, L., Burston, J. J., Kinsey, S. G., Schlosburg, J. E., et al. (2009). Selective blockade of 2-arachidonoylglycerol hydrolysis produces cannabinoid behavioral effects. Nature Chemical Biology, 5, 37–44.

25. Labar, G., Wouters, J., & Lambert, D. M. (2010). A review on the monoacylglycerol lipase: At the interface between fat and endocannabinoid signalling. Current Medicinal Chemistry, 17, 2588–2607.

26. Schlosburg, J. E., Blankman, J. L., Long, J. Z., Nomura, D. K., Pan, B., Kinsey, S. G., et al. (2010). Chronic monoacylglycerol lipase blockade causes functional antagonism of the endocannabinoid system. Nature Neuroscience, 13, 1113–1119.

27. Nomura, D. K., Lombardi, D. P., Chang, J. W., Niessen, S., Ward, A. M., Long, J. Z., et al. (2011). Monoacylglycerol lipase exerts dual control over endocannabinoid and fatty acid pathways to support prostate cancer. Chemistry & Biology, 18, 846–856.