Cot-death Vax SBS

Vaccines And Sudden Infant Death Syndrome

http://vaccineriskawareness.com

"Administration of Diphtheria-Petussis-Tetanus Toxoid (DPT) can cause temporary liver dysfunctions in infants similar to those resulting from viral hepatitis, and inoculation of killed Bordetella pertussis organisms makes some strains of mice 200 times more sensitive to histamine and three to five times as sensitive to endotoxins for approximately 14 days” - Am J. Dis Child 1955; 89:701-716 , http://archpedi.jamanetwork.com/article.aspx?articleid=498098#References

According to a Connaught vaccine manufacturer's data sheet for old style DPT vaccine,



'Sudden-infant-death-syndrome (SIDS) has been reported following administration of vaccines containing diphtheria and tetanus toxoids and pertussis vaccine. The significance of these reports is not clear. It should be borne in mind that the three primary immunizing doses of these vaccines are usually administered to infants between the age of 2 and 6 months and that approximately 85% of SIDS cases occur in the period from 1 through 6 months of age with the peak incidence at age 2 to 4 months.'



While they admit that SIDS has occured after vaccination with DPT containing vaccines, they attempt to brush it off by saying that that is because the majority of SIDS cases happen in the age range when vaccines are being given.



This isn't much of a defence, since it may be because of the shots that the SIDS occurs in that age group. Evidence suggests this to be the case (I'll discuss that further along in this article). Even the CDC admit the startling syncrinicity of the timing between SIDS and vaccines, but then discount it as 'coincidence' and say that studies show the two are not linked, while ignoring all the studies that say they are.



'From 2 to 4 months old, babies begin their primary course of vaccinations. This is also the peak age for sudden infant death syndrome (SIDS). The timing of these two events has led some people to believe they might be related.' - http://www.cdc.gov/vaccinesafety/Concerns/sids_faq.html



Later manufacturer's data sheets have become more mild in their interpretation of vaccine side-effects and many fail to mention SIDS, although they do admit to deaths after vaccination. According to Evans Medical, the Wellcome Foundation, product no. 0039/0474,



'Neurological events have occasionally been observed following the administration of pertussis-containing vaccines. The events reported do not appear to constitute a single, identifiable clinical syndrome but include isolated febrile convulsions, infantile spasms, episodes of persistent screaming and severe encephalopathy resulting in permanent brain damage or death. These events cannot be distinguished from those occurring in unvaccinated children of similar age. In the absence of a common, identifiable pathological mechanism, it is not possible to produce reliable estimates of the incidence of neurological events attributable to pertussis vaccination per Se.'



They state that severe screaming occurs, along with encepholopathy, permanent brain damage and death, yet attempt to brush that off by claiming unvaccinated babies have the same symptoms, which is pure nonsense.



1. Authorities have only ever done one double-blind controlled study on vaccination, in the 1970's, which showed more TB in the vaccine group. This type of study, which is the only type to get any answers of scientific value, has never been repeated. Doctors say that to withhold vaccines from children would be unethical, therefore they never study unvaccinated children.

'Unvaccinated' in their terms means children who have not had DPT containing vaccines, but have had other vaccines such as polio or Hepatitis B. It is known that ALL vaccines can cause screaming, encephalitis and death, so that would make it seem as if all babies are like that and it isn't down to the DPT.



2. Since they never study totally unvaccinated children, they cannot say that encephalitis and deaths occur in these groups.



3. Since the encephalitis, screaming and deaths occurs AFTER vaccination and not before, this surely is a clue that the vaccine had something to do with it. If a person became ill with something prior to any shots being given, one could obviously not blame vaccination. There would be another cause. But if the reoccuring factor is, the child has been vaccinated, began screaming, failed to eat and then died of 'SIDS' (as is the pattern many times over), then it is good medical practice to at least look at vaccination as a possible cause.



The medical profession and government bodies responsible for investigating SIDS are run by the Department of Health, and therefore they never look at vaccination as a possibility, even though the vaccines are being given when SIDS is peaking in incidence, when manufacturer's admit an association, when there have been dozens of journal papers suggesting a link and most importantly, when mothers are screaming

'My baby was vaccinated and he died. I know it wasn't SIDS.'

These mothers are ignored, told they are grieving, 'looking for someone to blame', or even worse, if the baby showed obvious vaccine injury such as retinal haemorraging, they are accused of causing the baby's death themselves. In recent years there has been clusters of parents accused of shaking their own children, like in the Sally Clark case where she lost one baby 24 hours after his DPT and another baby on the same day as his vaccinations. She served time for a crime she didn't commit and although later exonorated, found the trauma too much to bear and committed suicide.

Her third child, a boy, was not vaccinated during his infancy and he remains healthy.



Other possible causes are looked at: whether the parents smoke, what position the baby sleeps in, whether he has a dummy or not, if he's breast fed, whether he co-sleeps, even what his mattress is made out of. But they're not looking at the one immunological challenge that nearly all babies go through, vaccination.

Baby Deaths

Charlotte And Vance

Last year a Gloucester couple lost their baby daughter, Charlotte, a few days after her first vaccinations.



Charlotte, who was born on May 13 last year, had a combined jab for diphtheria, tetanus, whooping cough and polio, along with Hib meningitis and meningitis C vaccinations.



Carrie, 22, said: "Viral meningitis showed on Charlotte's autopsy but the final results came through as cot death.



"We're wondering if the vaccinations caused her death. She had them on Tuesday and she died the following Monday.



"More babies suffer from cot deaths in their first two to four months and that's when they have these injections."



Carrie and Dan live with Dan's father, David, who added: "It could have been a bad batch of vaccinations. We don't know what to believe - but they're not going to tell us."



Carrie found Charlotte dead in her cot at 7.30am on July 17.



She said: "The day before, Charlotte was fine. She'd drunk all her milk and had her daily feed before I put her to bed at the usual time - between 9pm and 10pm.



"Dan slept downstairs because Josie was in our bed. I found Charlotte the following morning. When I woke at 7.30am I thought, 'Hang on, she usually wakes up at 6am for her feed'.



"I noticed she had some blood coming from her nose. She wasn't moving or breathing and I screamed for Dan.



"We were trying to resuscitate her and I was screaming down the phone to the ambulance service asking, 'Where are the paramedics?'



"They took 15 minutes to arrive. We kept doing resuscitation but there was no sign of life.



"Deep down we knew she'd gone but didn't want to believe it.



"When the paramedics came we thought there could still be a chance, but they did resuscitation for half an hour and then rang the police."



Carrie denied smoking had affected her baby. She said: "If we had a fag we'd go into the kitchen and open a window or go outside. So did everyone else.



"We didn't want any of the children breathing in smoke and we didn't smoke in the car if the girls were there. We even had 'no smoking' signs on the door."



Charlotte wasn't planned but her parents were happy with the pregnancy.



Carrie said: "Everything was all right after the birth. She was a healthy, bubbly baby."



Dan added: "She was quiet and she was always smiling at you."



At the inquest, pathologist Dr Phillip Cox gave the cause of death as sudden infant death syndrome.



The inquest heard Charlotte had been born normally after a full-term pregnancy and had been developing well, according to a medical report into her death. (Taken from the Gloucestershire Echo, 15 January 2007).



Shelly Walker, from the USA, also lost her 4 month old son less than 3 days after his vaccinations. 2 other babies who were injected with the same batch of vaccine also died.



Shelly thinks it's no coincidence that her 4-month-old son died within days of receiving injections to prevent serious childhood illnesses, including diphtheria, tetanus, pertussis, hepatitis B, polio, rotavirus and invasive pneumococcal disease.



"My baby was so healthy," said Shelly Walker, 39, of Hayden. "He was extremely full of life, energy and vitality."



Nevertheless, early on the morning of Sept. 15, less than three days after Vance Vernon Walker received a round of vaccines at Lakeside Pediatric and Adolescent Medicine in Coeur d'Alene, his mother awoke to a nightmare.



"It was about 5:15 a.m. I woke up and thought, 'He's not making any noise!' " Walker recalled. "I went to pick him up and then I screamed."



Her 16 1/2-pound boy was warm and his lips were still pink, but he wasn't moving. Blood was crusted beneath his eyes, and his clothes and toys were covered with a bloody froth.



As her husband, Brian, 46, called 911, Walker worked frantically to resuscitate their child. But in the emergency room at Kootenai Medical Center, doctors said Vance had been dead for several hours.



"I grabbed my baby in my arms and held him up and I screamed, 'How in the hell did this happen?' " Walker said. "Was it the vaccines?"



Medical officials from the CDC and the federal Food and Drug Administration are working to answer that question for the Walkers and for families of two other babies who died within six weeks of each other.



Two of the deaths have been logged in the voluntary Vaccine Adverse Event Reporting System –VAERS – jointly operated by the CDC and FDA, agency officials said. But Dr. Robert West, the Kootenai County coroner, confirmed that three infants died this fall within days of immunization.



Parents of the other babies could not be reached for comment.



Autopsies failed to detect any specific vaccine reactions, West said, forcing a determination of SIDS – a "diagnosis of exclusion," he noted. (Taken from the spokesmansreview.com).

Two Month Baby Girl Dies

'Our beautiful daughter was born on February 14 and died on April 17. What was unusual was that earlier on the day she died I had taken her to the Military Base hospital for her two month checkup. The doctor told me that she was just perfect. Then the doctor said that she needed four shots. I replied Four!? She assured me that it was completely normal and that it was better to give her all at such an early age (because she wouldn't remember the shots). That evening after feeding [our daughter] we laid her down to sleep and checked on her 45 minutes later and discovered her dead. I told the police, coroner and investigators that I thought it was the shots because she was perfectly fine that day and before the shots. But after 3 weeks we finally got the answer from the autopsy that it was indeed SIDS. To this day I believe that it was the shots and no one can convince me otherwise.'



ANON.

Sabra Cline

Sabra has been given her first DPT shot on June 17 1985 in Tulsa, and over the next 48 hours the baby suffered a variety of puzzling symptoms: twitches and seizures, and a coolness to the skin, which also was turning blue

Mrs. Cline said she was aware of the mild symptoms which might be associated with a DPT shot, such as a mild fever and crankiness. But her baby wasn't experiencing those; these were much more frightening.

The infant was taken to a hospital in Muskogee, but Cline feels nothing could have been done by then to save her. "I was holding her when she died," the woman recalls.

At first, it was thought that Sabra had died from Sudden Infant Death Syndrome.

"I said 'No,' my baby's death was associated with the DPT shot. It had to be be," Cline says.

Official autopsy results released seven weeks later listed the cause of death as a swelling and inflammation of the brain tissue.

http://newsok.com/

SIDS IS NOT A DIAGNOSIS!!

You may have noticed in both of those cases, pathologists determined sudden infant death syndrome to be the cause of death.



SIDS is NOT a diagnosis! It is merely a term to describe a baby who has died suddenly when they don't know why. It is an UNEXPLAINED infant death. They cannot say 'SIDS was the cause of death', when they don't know what causes SIDS, so it may well have been a vaccination. According to the New England Journal of Medicine:



'In 1969, a National Institutes of Health consensus conference led to the first standardized definition of sudden infant death as the “sudden death of an infant or young child, which is unexpected by history, and in which a thorough post mortem examination fails to demonstrate an adequate cause of death.' (N Engl J Med. 2009 Aug 20; 361(8): 795–805. ).



Increasingly, more and more pathologists are using the terms SIDS on death certificates to explain away deaths they can't explain and it shouldn't be done. Any baby who has died suddenly despite being previously healthy, should be throughly investigated, including it's vaccination history.

Evidence That The Age Distribution Of SIDS Is Vaccine Related

So, is any of this more than just theory?



Between the years of 1970 and 1974 in Japan, there were 37 infant deaths directly after vaccination. Because of this, a group of doctors decided to boycott vaccines and vaccination was stopped altogether for two months. After that two months, the vaccination starting age was lifted to 2 years old, meaning that no child recieved a vaccine until after their 2nd birthday. Japan jumped from 17th place in child mortality to the lowest child mortality in the world. SIDS had disappeared.



In 1988 the vaccination age was lowered to 3 months old, and the SIDS rate rose again.



Cherry et al (1988), Pediatrics 81:6 Part 11 (June 1988) Supplement pp 936-984, said he found this to be 'instructive', suggesting a link between the Japanese vaccination schedule and SIDS.

Interestingly, Noble et al. (1987) who spent some 2 weeks in Japan studying the acellular whooping vaccine there, wrote: "It is difficult to exclude pertussis vaccines as a causal factor even when other etiologies are suggested, particularly when the adverse events occur in close temporal association with vaccination".



The same thing has happened in other countries when vaccination is stopped or delayed.



For instance, in the UK in 1975 and 1976, there was publicity about the whooping cough vaccine causing brain damage, so the vaccination rate fell to only 30%, and only 10% in some areas. In line with this, the infant mortality rate also fell.

By 1977, the controversy was starting to simmer down so vaccination rates began to climb up. And yes, you've guessed right, the infant mortality rate increased.



McFarlane (1982) wrote:



"The postneonatal mortality fell markedly in 1976, the year in which a sharp decline in perinatal mortality rate began. Between 1976 and 1979, however, neither the late nor the postneonatal mortality rates fell any further. Indeed, the postneonatal mortality rate increased ,slightly among babies born in 1977.'



This correlates exactly with the rise and fall of vaccination levels.



Fine and Clarkson (1982) wrote "...it is surprising that the interepidemic period did not decrease after the 1974 fall in vaccine uptake."

They were expecting unvaccinated children to come down with whooping cough in large numbers. Instead, they had the lowest ever recorded number of whooping cough hospitalisations and deaths.



More recently in India there has been a drop in infant mortality rates, also in line with decreasing vaccination:



Two recent health surveys carried out by the Government have thrown up mixed results. While one reports that the Infant Mortality Rate has fallen below 60 for the first time in the country, the worrying sign is that the already low immunisation rates are showing further decline.



The most alarming is the case of Uttar Pradesh, which shows a fall in immunisation from 43.7 per cent in 1998-99 to 28.1 per cent in the latest data.



In 1998-1999, 54 per cent of the children in the country were reported to be fully immunised. But a district household survey 2002-2004, the data for which was released last month, shows a decline in this to 47.6 per cent. In 1989-99, India had one-third of the world?s non-immunised children.



Immunisation rates seem to have fallen across the country, including Uttar Pradesh and Bihar, which account for 40 per cent of the total children in the age group of zero to one who need immunisation. But unlike Uttar Pradesh, Bihar has shown only a marginal decline, from 24.4 to 22.4 per cent.



Experts believe that the focus on polio eradication, at the cost of routine immunisation, could have contributed to the decline.



The other states showing low figures are Rajasthan (25.4 per cent), Tripura (26.7 per cent), Jharkhand (29.3 per cent) and Madhya Pradesh (32.5 per cent).



The states at the other end of the spectrum are Tamil Nadu (with an immunisation rate of 92.1 per cent), Kerala (81.2 per cent), Pondicherry (89.4 per cent), Goa (81.5 per cent) and Himachal Pradesh (79.4 per cent).



There is good news, however, on the infant mortality front. For the first time, India has reported IMR below 60, with the survey from Registrar General of India released recently showing 58 deaths per 1,000 live births in the country.



Though the rates are still high compared to other countries, the figures have shown decline from 68/1,000 live births in 2000, and 60/1,000 live births in 2004.' (Indian Express).



i.e. Vaccination rates FELL from 54% in 1999 to 47.6% now and the infant mortality rate has fallen from 68 per 1000 live births in 2000, to 58 per 1000 now.



I'd say that's clear evidence of a link between childhood vaccination and infant mortality.

Medical Research Into Vaccines and SIDS

A study in Pediatr Infect Dis. 1983 Jan-Feb;2(1):7-11. PMID: 6835859; UI: 83169234, was to determine whether or not DPT caused SIDS. They contacted the parents of 145 SIDS victims who died in LA between January 1979 and August 1980. Of these, 53 babies had had DPT vaccination. Of those 53, 27 had been given a DPT shot within 28 days of death, 17 deaths occured within 1 week of the vaccination and 6 deaths within 24 hours. This was significantly more than expected and points to a temporal relationship between DPT and SIDS.



In addition to this there were also 46 babies who died who didn't have DPT but had visited a clinic before they died (possibly for other vaccines?). 40 of them died within 28 days of the visit, 22 a week after the visit and seven on the third day after the visit.



However, the researchers refused to have a control group (i.e. babies who died of SIDS who had recieved NO vaccines), because they said

'There should be no temporal association between DPT immunization and SIDS.'



So they were never able to get a truly accurate picture, as is the case with all vaccine 'science'.



Other studies include:



Torch, W.S., 1982. Diphtheria-pertussis-tetanus (DPT) immunization: a potential cause of the Sudden Infant Death Syndrome (SIDS). Neurology; 32(4): A169 abstract).



Torch, W.C., 1986 a. Characteristics of diphtheria-pertussis-tetanus (DPT) postvaccinal deaths and DPT-caused Sudden Infant Deaths Syndrome (SIDS): a review. Neurology (suppl 1); 36: 148 (abstract).



Torch, W.C., 1986 b. Diphtheria-pertussis-tetanus (DPT) imunization may be an unrecognized cause of Sudden Infant Death (SIDS) and Near-Miss Syndrome (NMS): 12 case reports. Neurology (suppl 1); 36: 149 (abstract).



Cherry, J.D. (1988), Brunell, P.A., Golden, G.S., Karzon, D.T., (1988), Report of the task force on pertussis and pertussis immunization, Pediatrics 81:6 Part 11 (June 1988) Supplement pp 936-984.

Excerpt: The rate of severe reactions does not differ significantly between the acellular and whole-cell vaccines when used at 24 months of age. The decrease in severe reactions is slight, if any. The category "sudden death" is also instructive in that the entity disappeared following both whole-cell and acellular vaccines when immunisation was delayed until a child was 24 months of age. It is clear that delaying the initial vaccination until a child is 24 months, regardless of the type of vaccine, reduces most of the temporally associated severe adverse events.



Torch, one of the researchers into DPT vaccine and crib death, reported at the 34th annual meeting of the American Academy of Neurology that:



'DPT may be a generally unrecognized major cause of sudden infant and early childhood death, and that the risks of immunization may outweigh its potential benefits. A need for re-evaluation and possible modification of current vaccination procedures is indicated by this study.'



(Torch WC. Diphtheria-pertussis-tetanus (DPT) immunization: a potential cause of the sudden infant death syndrome (SIDS). American Academy of Neurology, 34th Annual Meeting, Apr 25-May 1, 1982. Neurology 32(4): pt. 2).



In Current Medicinal Chemistry, although researchers didn't find a causal link, they did say:



'We selected 110 cases submitted to in-depth histological examination of the autonomic nervous system and provided with detailed clinical and environmental information. In 13 cases (11.8%) the death occurred in temporal association with administration of the hexavalent vaccine (from 1 to 7 days). In none of these victims congenital developmental alterations of the main nervous structures regulating the vital functions were observed. Only the hypoplasia of the arcuate nucleus was present in 5 cases. In one case in particular an acquired hyperacute encephalitis of the tractus solitarii nucleus was diagnosed in the brainstem.....we hypothesize that vaccine components could have a direct role in sparking off a lethal outcome in vulnerable babies.'



Source: Current Medicinal Chemistry, Volume 21, issue 7, DOI 10.2174/09298673113206660289





Sudden Infant Death Following Hexavalent Vaccination: A Neuropathologic Study

Abstract:

We examined a large number of sudden infant death syndrome victims in order to point out a possible causal relationship between a previous hexavalent vaccination and the sudden infant death. We selected 110 cases submitted to in-depth histological examination of the autonomic nervous system and provided with detailed clinical and environmental information. In 13 cases (11.8%) the death occurred in temporal association with administration of the hexavalent vaccine (from 1 to 7 days). In none of these victims congenital developmental alterations of the main nervous structures regulating the vital functions were observed. Only the hypoplasia of the arcuate nucleus was present in 5 cases. In one case in particular an acquired hyperacute encephalitis of the tractus solitarii nucleus was diagnosed in the brainstem. This study does not prove a causal relationship between the hexavalent vaccination and SIDS. However, we hypothesize that vaccine components could have a direct role in sparking off a lethal outcome in vulnerable babies. In conclusion, we sustain the need that deaths occurring in a short space of time after hexavalent vaccination are appropriately investigated and submitted to a post-mortem examination particularly of the autonomic nervous system by an expert pathologist to objectively evaluate the possible causative role of the vaccine in SIDS.



'We hypothesize that vaccine components could have a direct role in sparking off a lethal outcome in vulnerable babies.' Source: DOI: 10.2174/09298673113206660289





How Would Vaccines Cause SIDS?

All of the ingredients in vaccinations are poisons and information on them can be found at a poison's unit or environmental health and safety. When they are contained in other products, we hear how dangerous they are to our health, for instance, glycol in paint has been attributed to male infertility, yet this is in vaccines. Mercury was banned from the fillings of pregnant women because of it's danger, formaldehyde is a known trigger of throat cancer and everyone knows that aluminium can make you lose your memory.



If you took a single ingredient like this and put it into a baby, I guarantee you'd have at least one baby who would die as a result. With vaccines, their tiny little bodies also have to cope with simultaneous viruses and bacteria, which would never happen in nature, and if you consider that at birth a baby doesn't really have his own immune system. He is protected by placental antibodies and his mother's milk. A child's immune system is not fully developed until the age of 6 years. Just like tiny babies who are at greater risk of dying of the natural diseases, some babies cannot cope with the vaccine toxins and just like with any poisoning death, it doesn't always happen instantly. The body's vital organs can shut down over time.

The Mechanisms of A Vaccine SIDS Death

CYTOKINE STORM



Cytokines are pro-inflammatory messengers that instruct T cells and macrophages to travel to a site of infection (thus creating inflammation). This is a mechanism of the immune system to kill off viruses and bacteria and to repair injury. However, sometimes too many cytokines are formed in a hyper-immune response and this can cause massive inflammation and tissue damage. Depending on where the T cells and macrophages are directed to, it can also cause respiratory failure due to swelling blocking the airway. It can also cause sepsis and septic shock.



This is called cytokine storm. This has occured after vaccination and infection as a hyper-immune response.

SIDS cases have increased levels of interleukin-6 in cerebrospinal fluid.

Abstract

Cerebrospinal fluid (CSF) from 20 infants who died of sudden infant death syndrome (SIDS), 7 cases of infectious death and 5 cases of violent death were examined with respect to concentrations of interleukin-6 (IL-6). The measurements were performed by ELISA. IL-6 levels in SIDS were significantly lower than in infectious death (p < 0.02), but significantly higher than in violent death (p < 0.02). Since IL-6 plays an important role in immune responses and may induce fever, the findings may suggest that immune activation plays a role in SIDS. The presence of cytokines in the central nervous system (CNS) may cause respiratory depression, especially in vulnerable infants.



Source: Acta Paediatr. 1995 Feb;84(2):193-6.

Long peptide vaccination can lead to lethality through CD4+ T cell-mediated cytokine storm.

Abstract

The optimization of anticancer therapeutic vaccines can lead to better efficacy but also to stronger adverse effects. In a mouse model of antitumor vaccination using a long peptide (LP), which included MHC class I- and II-restricted male (H-Y) epitopes, we observed unexpected mortality. Mice with an increased frequency of anti-H-Y CD4 T cells were primed with LP+CpG and boosted 10 d later. Within hours of boost, they displayed shock-like signs with high mortality. Serum cytokine levels were high. TNF-alpha secreted by the CD4 T cells was identified as the key effector molecule. Priming with a short peptide (SP), which included the MHC class II-restricted epitope, was a more efficient primer than LP, but did not lead to mortality when used as boost. The high mortality induced by LP compared with SP was probably related to its specific ability to be presented by B cells. Finally, targeting the LP sequence to dendritic cells allowed tumor protection without side effects. Our data: 1) confirm that the immune system can be very dangerous; 2) caution against the use of systemic activation of high-frequency Ag-specific T cells as induced by high doses of LP; and 3) underline the benefit of targeting Ag to dendritic cells.



Source: J Immunol. 2010 Jul 15;185(2):892-901. doi: 10.4049/jimmunol.1000933. Epub 2010 Jun 11.

Interleukin-2 as a neuromodulator possibly implicated in the physiopathology of sudden infant death syndrome.

Abstract

Dysfunction in vital brainstem centers, including those controlling cardiorespiratory- and sleep/arousal pathophysiology, is reported in sudden infant death syndrome (SIDS). Biological mechanisms underlying SIDS, however, remain unclear. Cytokines are inter-cellular signaling chemicals. They can interact with neurotransmitters and might thus modify neural and neuroimmune functions. Cytokines could therefore act as neuromodulators. Interleukin (IL)-2 is a major immune-related cytokine. It has not been previously depicted in vital brainstem centers. We detected intense neuronal IL-2 immune-reactivity in the SIDS brainstem, namely in vital neural centers. This IL-2 overexpression might interfere with neurotransmitters in those critical brainstem centers, causing disturbed homeostatic control of cardiorespiratory and arousal responses, possibly leading to SIDS.



Source: Neurosci Lett. 2010 Aug 16;480(2):122-6. doi: 10.1016/j.neulet.2010.06.021. Epub 2010 Jun 11.



Acute Necrotizing Encephalopathy: An Underrecognized Clinicoradiologic Disorder



'ANE may also develop secondary to diphtheria, tetanus toxoid, and whole-cell pertussis (DPTw) vaccination [26]. The DPTw vaccination might result in increased production of cytokines and cause the alteration of vessel wall permeability, leading to local breakdown of BBB [38, 44–47].' (BBB is blood brain barrier).



Source: Mediators of Inflammation

Volume 2015 (2015), Article ID 792578, 10 pages

http://dx.doi.org/10.1155/2015/792578

Possible pathomechanisms of sudden infant death syndrome: key role of chronic hypoxia, infection/inflammation states, cytokine irregularities, and metabolic trauma in genetically predisposed infants.

Abstract

Chronic hypoxia, viral infections/bacterial toxins, inflammation states, biochemical disorders, and genetic abnormalities are the most likely trigger of sudden infant death syndrome (SIDS). Autopsy studies have shown increased pulmonary density of macrophages and markedly more eosinophils in the lungs accompanied by increased T and B lymphocytes. The elevated levels of immunoglobulins, about 20% more muscle in the pulmonary arteries, increased airway smooth muscle cells, and increased fetal hemoglobin and erythropoietin are evidence of chronic hypoxia before death. Other abnormal findings included mucosal immune stimulation of the tracheal wall, duodenal mucosa, and palatine tonsils, and circulating interferon. Low normal or higher blood levels of cortisol often with petechiae on intrathoracic organs, depleted maternal IgG antibodies to endotoxin core (EndoCAb) and early IgM EndoCAb triggered, partial deletions of the C4 gene, and frequent IL-10-592*A polymorphism in SIDS victims as well as possible hypoxia-induced decreased production of antiinflammatory, antiimmune, and antifibrotic cytokine IL-10, may be responsible for the excessive reactions to otherwise harmless infections. In SIDS infants, during chronic hypoxia and times of infection/inflammation, several proinflammatory cytokines are released in large quantities, sometimes also representing a potential source of tissue damage if their production is not sufficiently well controlled, eg, by pituitary adenylate cyclase-activating polypeptide (PACAP) and vasoactive intestinal polypeptide (VIP). These proinflammatory cytokines down-regulate gene expression of major cytochrome P-450 and/or other enzymes with the specific effects on mRNA levels, protein expression, and enzyme activity, thus affecting metabolism of several endogenous lipophilic substances, such as steroids, lipid-soluble vitamins, prostaglandins, leukotrienes, thromboxanes, and exogenous substances. In SIDS victims, chronic hypoxia, TNF-alpha and other inflammatory cytokines, and arachidonic acid (AA) as well as n-3 polyunsaturated fatty acids (FA), stimulated and/or augmented superoxide generation by polymorphonuclear leukocytes, which contributed to tissue damage. Chronic hypoxia, increased amounts of nonheme iron in the liver and adrenals of these infants, enhanced activity of CYP2C9 regarded as the functional source of reactive oxygen species (ROS) in some endothelial cells, and nicotine accumulation in tissues also intensified production of ROS. These increased quantities of proinflammatory cytokines, ROS, AA, and nitric oxide (NO) also resulted in suppression of many CYP450 and other enzymes, eg, phosphoenolpyruvate carboxykinase (PEPCK), an enzyme important in the metabolism of FA during gluconeogenesis and glyceroneogenesis. PEPCK deficit found in SIDS infants (caused also by vitamin A deficiency) and eventually enhanced by PACAP lipolysis of adipocyte triglycerides resulted in an increased FA level in blood because of their impaired reesterification to triacylglycerol in adipocytes. In turn, the overproduction and release of FA into the blood of SIDS victims could lead to the metabolic syndrome and an early phase of type 2 diabetes. This is probably the reason for the secondary overexpression of the hepatic CYP2C8/9 content and activity reported in SIDS infants, which intensified AA metabolism. Pulmonary edema and petechial hemorrhages often present in SIDS victims may be the result of the vascular leak syndrome caused by IL-2 and IFN-alpha. Chronic hypoxia with the release of proinflammatory mediators IL-1alpha, IL-1beta and IL-6, and overloading of the cardiovascular and respiratory systems due to the narrowing airways and small pulmonary arteries of these children could also contribute to the development of these abnormalities. Moreover, chronic hypoxia of SIDS infants induced also production of hypoxia-inducible factor 1alpha (HIF-1alpha), which stimulated synthesis and release of different growth factors by vascular endothelial cells and intensified subclinical inflammatory reactions in the central nervous system, perhaps potentiated also by PACAP and VIP gene mutations. These processes could lead to the development of brainstem gliosis and disorders in the release of neuromediators important for physiologic sleep regulation. All these changes as well as eventual PACAP abnormalities could result in disturbed homeostatic control of the cardiovascular and respiratory responses of SIDS victims, which, combined with the nicotine effects and metabolic trauma, finally lead to death in these often genetically predisposed children.



Source: Am J Ther. 2004 Nov-Dec;11(6):517-46.

Urinary tract diseases revealed after DTP vaccination in infants and young children: cytokine irregularities and down-regulation of cytochrome P-450 enzymes induced by the vaccine may uncover latent diseases in genetically predisposed subjects.

Abstract

Prophylactic vaccinations may sometimes shorten the incubation period of some illnesses and/or convert a latent infection/inflammation into a clinically apparent disease. Cytokines play a major role in mediating the inflammatory process in various clinical entities and represent a potential source of tissue damage if their production is not sufficiently well controlled. It seems that irregularities in production of proinflammatory cytokines may be responsible for the abnormalities associated with full-blown clinical symptoms of various urinary tract diseases observed after DTP vaccination in 13 infants and young children hospitalized over the past 24 years. On admission, upper respiratory tract diseases, atopic dermatitis, and/or latent urinary tract infection/inflammation were found in these children. It is suggested that the whole-cell pertussis present in DTP vaccine, acting as an excessive stimulus in these patients, produced symptoms reminiscent of biologic responses to circulating proinflammatory monokines such as IL-1beta, TNF-alpha, and IL-6 because earlier it was reported that in vitro the whole-cell vaccine induced significantly more such cytokine production than did the acellular pertussis or diphtheria-tetanus-only vaccine. Analysis of the cellular immune disturbances previously reported in urinary tract infection/inflammation (increased serum and/or urinary IL-1alpha, IL-1 receptor antagonist, IL-6 and IL-8), steroid-sensitive nephrotic syndrome (increased IL-2, IFN-gamma, TNF-alpha, and decreased or increased IL-4, depending on the cells studied), and atopic dermatitis (decreased IFN-gamma and increased IL-4 production), may suggest that similar subclinical chronic cytokine-mediated abnormalities produced in the course of latent diseases revealed in our patients, combined with those caused by DTP vaccination stimulus, were responsible for the pathomechanism of these clinical entities. This speculation is in agreement with the reports on the long-lasting induction of cytokine release and down-regulation of hepatic cytochrome P-450 isoenzyme activities after administration of DTP vaccine to mice and may be supported by the fact that TH1 phenotype is associated with the up-regulation of intercellular adhesion molecule-1 and RANTES, whereas TH2 phenotype is associated with the up-regulation of the vascular cell adhesion molecule and P-selectin, which are key players in the migration into inflamed tissues and localization of lymphocytes and other allergic effector and inflammatory cells. Because several inflammatory cytokines down-regulate gene expression of major cytochrome P-450 and/or other enzymes with the specific effects on mRNA levels, protein expression, and enzyme activity, thus affecting the metabolism of several endogenous lipophilic substances such as steroids, lipid-soluble vitamins, prostaglandins, leukotrienes, thromboxanes, and exogenous substances, their irregularities in the body may eventually lead to the flare of latent diseases in some predisposed subjects. Also, interleukin genetic polymorphisms, especially the constellation of TNF-alpha and IL-6 genetic variants, might predispose some infants with infection to a more than usually intense inflammatory response in the kidneys after vaccination. It seems that the aforementioned pathomechanism may also be responsible for some cases of sudden infant death syndrome, which is often preceded by infection/inflammation.





Source:

Am J Ther. 2004 Sep-Oct;11(5):344-53.

Stress

There are several ways in which a baby could die after a vaccination. They are:



1. Stress. The stress on the body of all the viruses and added toxins, plus the pain of the injections can cause altered heart rate and breathing patterns. It is well known that stress can cause heart disease in adults. I firmly believe that stress can affect babies and children, too. A recent study I read showed that babies who have a dummy at night, don't seem to die of SIDS. Why, well, sucking on something comforts them so reduces their adreneline levels and helps regulate breathing. Of course it would be better if they were suckling from mum, but these days where most babies are put to bed separately, I can see how a dummy would reduce stress levels and associated SIDS.



Viera Scheibner is a research scientist who developed the Cotwatch monitor with her late husband, and she found that baby's breathing patterns were stressed after vaccination:



'Scientific evidence shows that babies can have severe adverse reactions to vaccinations at critical intervals following their shots, and that vaccination is the more likely cause of cot death and shaken baby syndrome.



Dynamics of critical days as part of the dynamics of non-specific stress syndrome discovered during monitoring with Cotwatch breathing monitor.



Cotwatch was a true breathing monitor, meaning its electronics separated heartbeat and breathing and only breathing delayed the alarm. The feedback on breathing from the standard home monitor was from alarms (figure 1), while the microprocessor-based unit provided computer printouts of the record of breathing in the form of histograms stacked up at an angle (figure 2) or vertical bars (figures 3,4), the length of which directly reflected the stress level as integrals of the weighted apnoea/hypopnoea density (WAHD).



The record of alarms in a baby over a period of five-and-a-half months, from October 1987 to March 1988 (figure 1), reveals that the stress-induced breathing pattern did not subside after 21 days following vaccine administration: it was still continuing on and off (following the critical days) two-and-a-half to three months later; and before really recovering from the first lot, the child was given the second injection of DPT and oral polio vaccines. Cotwatch recorded events in breathing: apnoeas (pauses in breathing) and hypopnoeas (low-volume breathing, i.e., below 5% of the volume of normal unstressed breathing). The events were logarithmically weighted (the figures on the vertical axis of the computer printouts are integrals of the WAHD).



The first two charts in figure 3 are computer printouts of the record of events in breath­ing in two babies: baby one, who was given the third DPT (diphtheria-pertussis-tetanus) vaccine and OPV (oral polio vaccine); and baby two, who was given the first DPT and OPV. The higher the vertical bar, the higher the stress level in breathing; figure 3 shows flare-ups of stress-induced breathing day by day from day 0 when the vaccines were administered and up to the 17th day.



It is obvious that even though baby one reacted much more than baby two, the flare-ups of stressed breathing followed the same pattern of critical days, the most important of these being day 2, after which day the stress level went down and started rising again between days 5 and 7, when the stress level subsided and started increasing again between days 14-16, subsided again and rose again between days 19-24, after which it subsided and rose again towards the 28th day and so on, following closely the pattern of alarms as recorded by a mother of one baby (figure 1). Days 10 or 11 also emerged as critical days in babies who reacted strongly, such as baby one. Needless to say, the increased intensity of reactions after the third DPT injection and OPV reflects the phenomenon of sensitisation (sensitisation in this context means increased deranged immunological response or anaphylaxis; and in the case of vaccines it also means increased susceptibility to the dis­eases that the vaccines are supposed to prevent and to a host of unrelated bacterial and viral infections (Parfentjev, 1955; Craighead, 1975; Daum et al., 1989).



The third chart in figure 3 is of 41 actual, randomly listed deaths after DPT and OPV; it can be seen that the distribution of deaths closely follows the dynamics of the flare-ups of stressed breathing of babies one and two after the administration of the DPT vaccine (Bernier et al., 1982, Walker et al., 1987, Coulter and Fisher, 1991).



Generally speaking, the most fundamental error of judgement displayed by cot death researchers is that they do not look at what happened to the babies who succumbed to SIDS, days before they died, and instead they try to identify the elusive entity of "at risk" babies. The pneumographic studies are done without any regard to what happens to babies in the first six months and/or one year or 18 months of life when the initial DPT, Hib and polio vaccines and the first MMR and/or booster vaccines are given.



Vaccinations are mostly ignored in cot death studies. In our experience, the timing of pneumographic studies is determined by the availability of a bed in the overnight study unit rather than by looking at what happened to the baby just before it developed symptoms of stress or started causing alarms on its monitor.



The notion of false alarms, widely used by those who conduct monitoring of babies' breathing, has also delayed the understanding of the situation. Alarms which occurred when the monitored baby did not stop breathing but was breathing very shallowly are considered false alarms. Leif and I called them "warning alarms" because they sounded when the monitored babies started having longer and longer episodes of low-volume breathing, which is the true stress-induced breathing pattern. A baby who developed pneumonia experienced such "false alarms" for two weeks before going down with typical symptoms of pneumonia. This happened about six weeks after the six-month vaccination with DPT and polio vaccines.



When reactions or deaths occur six weeks after vaccination, they would not be considered as being caused by vaccination. Yet our records of alarms with Cotwatch micro­processor computer printouts demonstrate increased stress level in breathing more than six weeks after vaccination. - Viera Scheibner, 'Vaccination and the dynamics of critical days.'

Immediate Shock

Sometimes a baby can have a shock reaction to a vaccine only minutes or hours after it is given, with difficulty breathing, brain swelling (encephalitis) and blood coming from the eyes or mouth. This happens more frequently in the third world where they often use vaccines which have been banned in Western countries for safety reasons. (See 'Killer Measles Vaccine is Withdrawn' page).

Immune Paralysis

Vaccines challenge the immune system, usually too much, so in the days after vaccination, when the body is trying to fight the viruses and heavy metals, it can actually make you more suseptible to the diseases or even CAUSE the disease.



According to a Hibtiter manufacturer's data sheet from Lederle Laboratories, 1999,



'As with any vaccine, HibTITER may not protect 100% of individuals receiving

the vaccine. Also, as reported with other vaccines, cases of Haemophilus B

disease may occur prior to the onset of the protective effects of the

vaccine.'



And:



'Two children developed Haemophilus B disease after receiving the

two dose primary vaccination schedule. One child became ill at 15 months of

age and the other at 18 months of age....There have been rare reports to the Vaccine Event Reporting

System (VAERS) of Haemophilus B disease following full primary immunisation.'



In the days or weeks after vaccination, the baby may get HIB disease, blood poisoning, meningitis, pneumonia and succumb that way, or be left with lots of respiratory infections as are common with vaccinated children, and then eventually succumb to SIDS. This is because their immune system has been weakened and foreign bacteria introduced to the body, so they are unable to stay healthy.



Dr. Harold Buttram said

'As reported in the New England Journal of Medicine in 1984 [23], 11 healthy adults had tests involving subpopulations of T-lymphocytes (white blood cells which mediate immune function) before and after routine tetanus booster immunizations. The results showed a significant though temporary drop in T-helper lymphocytes. Special concern rests in the fact that in 4 of the subjects the T-helper lymphocytes dropped to levels found in active AIDS patients. If this was the result of a single vaccine in healthy adults, it is sobering to think of the immune consequences of the multiple vaccines given to infants with their immature and vulnerable immunity. This study should have served as a pilot for ongoing vaccine safety studies, but as far as can be determined from surveys of the medical literature, it has never been repeated.'

Vaccine Induced Scurvy

You've all heard of scurvy, the sailor's disease caused by malnutrition, well, it may surprise you to learn that vaccines can cause exactly that.



How?



Dr. Archie Kalokerino's worked as a physician in Australia with aboriginal people and after a DPT 'immunisation' campaign, he noticed to his horror that 50% - yes HALF - of all babies were dying. After researching what may be the cause of this huge death rate, he realised that the majority of these babies were vitamin C deficient and were unable to cope with the vaccine.



Dr. Thomas Levy writes:



'Vaccinations also generally present some degree of toxin insult to the body. Kalokerinos (1981) observed that vitamin C-deficient Aboriginal infants were often placed into an acute state of scurvy because of the additional vitamin C demands placed on their bodies by the vaccination injections, resulting in sudden death. (Thomas E. Levy, MD, JD, Vitamin C, Infectious Diseases, & Toxins – Curing The Incurable, 2002).'



After supplementing these babies with vitamin C he found that they no longer died after vaccination. He was awarded the Nobel Peace Prize for his work.



So how can subclinical scurvy kill?



Vaccination is a major assault to the system and it requires many vitamins to use in the production of more antibodies (see 'How Your Immune System Works' page). If there aren't enough vitamins present, then the immune system will leach them from bones and other vital organs to fight the infection. This can cause internal bleeding (and subsequent bruising), fractures of the bones and haemorraging of the retinas and other areas. All of these complications can lead to death.

It is why some SIDS babies have been found with blood on their mouth or clothing.

Sometimes the medical profession will accuse the parents of child abuse in an attempt to explain away the child's injuries, a scenario which is happening more frequently now that there are more childhood vaccines and more children dying.



According to an article by Jonathan Campbell,



'The "Recommended Daily Allowance" of vitamin C is a small fraction of the amount we - and infants - really need. Many metabolic processes will be compromised, but the outward signs won't be obvious. Artery wall repair will happen more slowly, and the human body compensates for this deficiency with a sticky plaque called lipoprotein(a) - the root cause of cardiovascular disease. Antibodies and white blood cells will be built incorrectly or not at all. Cholesterol, needed for nutrient transport, will not be recycled properly.







For infants, this is deadly. Their little bodies have very little reserves to draw upon. Without sufficient vitamin C, their immune systems and arteries are fragile. A single stressful event, a minor fall, a vaccination, a toxic exposure, or a simple virus or bacterial illness could tip the balance and kill them. Metabolic failure, heart failure, toxic trauma to vital organs, hemorrhage - it could happen dozens of ways. Sudden death, with no warning. SIDS.

For vaccinations: In general, I recommend that parents seriously consider not vaccinating their children, and certainly never to vaccinate against hepatitis B, because the vaccine has a horrific reputation for harming children, and hepatitis B is both extremely rare and also quite curable. Vaccines in general contain both live (but "attenuated") viruses and a brew of toxic chemicals and preservatives, sometimes including mercury (thimerosal). This places a huge burden on the child's immune system, and quickly depletes vitamin C. Many SIDS victims have died shortly after vaccinations.







If you decide to vaccinate your child, increase the vitamin C dose dramatically several hours before and for several days after the vaccination to prevent vitamin C depletion and so that the child's immune and detoxification systems will have a chance to kill the viruses and neutralize the toxins. Demand non-thimerosal, single-dose, single-virus vaccines; the multiple-virus vaccines such as DPT and MMP have the worst reputations regarding harm to infants.'



Vaccines and vitamin C deficency are discussed in this medical paper:



Hattersley J, The Answer to Crib Death “Sudden Infant Death Syndrome” (SIDS), Journal of Orthomolecular Medicine Volume 8, Number 4, 1993, pp.229-245



'Abstract



(1) Two doctors on opposite sides of the globe eliminated crib death among their patient populations for 40 years using ascorbate supplementation. Unknown to each other they arrived at the same regimen.



(2) Crib deaths nearly disappeared in Japan in 1975 when first inoculations were postponed until the 24th month of life.



These findings and their explanation are explored. SID is traced to a nonspecific or general adaptation stress syndrome defined by Hans Selye. It is precipitated by a deficiency of ascorbate and also of vitamin B6 and zinc.'





Conclusion

To minimize the risk of SIDS for your baby:



1. Breast feed (this will be discussed on another page).



2. Consider not vaccinating.



3. If you do decide to vaccinate, consider delaying them until your child is 2.



4. If you do decide to vaccinate, supplement your child with vitamin C before and after the vaccines.



5. If you do decide to vaccinate, make sure the shots don't have thimerosal (mercury) as an excipient. Space out the jabs and avoid multiple combinations.



6. Even if your child is unvaccinated, if he is sick with anything, was pre-term or is formula fed, consider giving vitamin C supplementation.

Sudden Unexpected Death in Childhood

Sudden Unexpected Deaths and Vaccinations during the First Two Years of Life in Italy: A Case Series Study

As more and more vaccines have been added to the schedule and more booster doses given, there are an increasing number of children dying suddenly when they are over the age of one year. This presented a problem for the medical community, whose definition of SIDS was a sudden, unexpected death with no known cause any time during the first year of life. How then could they explain how previously healthy children over the age of one, just suddenly dropped dead?



They invented a new term, Sudden Unexpected Death in Childhood, in an attempt to explain this away.



Ocasionally a child is found to have an underlying medical problem that was not detected during life, but for some children, research shows that their death is down to vaccines.

Background



The signal of an association between vaccination in the second year of life with a hexavalent vaccine and sudden unexpected deaths (SUD) in the two days following vaccination was reported in Germany in 2003. A study to establish whether the immunisation with hexavalent vaccines increased the short term risk of SUD in infants was conducted in Italy.



Methodology/Principal Findings



The reference population comprises around 3 million infants vaccinated in Italy in the study period 1999–2004 (1.5 million received hexavalent vaccines). Events of SUD in infants aged 1–23 months were identified through the death certificates. Vaccination history was retrieved from immunisation registries. Association between immunisation and death was assessed adopting a case series design focusing on the risk periods 0–1, 0–7, and 0–14 days after immunisation. Among the 604 infants who died of SUD, 244 (40%) had received at least one vaccination. Four deaths occurred within two days from vaccination with the hexavalent vaccines (RR = 1.5; 95% CI 0.6 to 4.2). The RRs for the risk periods 0–7 and 0–14 were 2.0 (95% CI 1.2 to 3.5) and 1.5 (95% CI 0.9 to 2.4). The increased risk was limited to the first dose (RR = 2.2; 95% CI 1.1 to 4.4), whereas no increase was observed for the second and third doses combined.



Source: Traversa G, Spila-Alegiani S, Bianchi C, Ciofi degli Atti M, Frova L, et al. (2011) Sudden Unexpected Deaths and Vaccinations during the First Two Years of Life in Italy: A Case Series Study. PLoS ONE 6(1): e16363. doi:10.1371/journal.pone.0016363

Dr. David Davis MD, Talks about the Link Between Vaccines, PVC and Crib Death

"My suspicion, which is shared by others in my profession, is that the nearly 10,000 SIDS deaths that occur in the US each year are related to one or more of the vaccines that are routinely given to children. The pertussis (whooping cough) vaccine is the

most likely villain , but it could also be one or more of the others. " Dr. R. Mendelsohn - Pediatrician.

Doctor’s Son Dies of SIDS After Vaccines



Nash Brandon Perry was born on 10/26/2015 and taken from us on 1/17/16. Nash was put down on his back for a nap, when I checked on him about an hour later I found my son face down, arms straight down at his sides and legs straight out. I knew something was wrong. I ran, picked him up and immediately saw the horror that was my precious baby boy dead, blue in the face, with blood coming out of his nose. I attempted CPR until EMS arrived but it was too late.

Now, maybe it is my grief just looking for answers or something to blame, but there are so many things that just don’t add up. My son never rolled over except twice while throwing a fit and throwing himself around, never while sleeping. I was one room away, he never screamed or cried. Although he was only 2.5 months he had a strong neck. He could hold it up and he could definitely turn it to the side. He had slept the same way many times with no problems. He had no underlying medical problems. My research has found dozens of similar, horrible stories from other parents.

Now, I could just write it off as God’s will or bad luck. But like I said, I’m a scientist, I’m a doctor. There are some very disconcerning similarities between my son’s death and a lot of the SIDS deaths I have read about. A lot of those people were told, “well that’s just coincidence because the highest incidence of SIDS is between 2 and 6 months.” But what if it’s not coincidence? What if we (doctors) are doing routine things to our babies that put them at higher risk that could easily be adjusted to lower the risk, i.e. not giving 8-12 vaccines at one doctors visit to infants. What if Nash, and some other SIDS victims, had some predisposition that made them susceptible to reacting badly to that much vaccine at one time. I am in no way anti-vaccine because they have saved millions of lives, but what if giving them the way we do is contributing to SIDS (our son had his days before). This is just one hypothesis that I want to be able to research. I also want to reach out to other victim’s families, listen to their stories and their hypotheses. During my time as a doctor, I’ve found if you ask the right questions and actually listen to the patient a lot of times they know exactly what is going on or at least have come up with good theories.

What we are asking of you all, those who have shown desire to send us flowers or gifts, instead donate a little money to my new journey. I’m a doctor and now I know my purpose. My purpose is to search for causal relationships between things we are doing that are putting babies at risk of SIDS. To make sure fewer and fewer families have to suffer this horrible tragedy that we are going through . I live in Bexar County which has the highest SIDS rates in all of Texas, so I am confident I am in the right place to gather great data and do good research.

Money donated will be used to fund my research and any money left over will be invested in the companies that are making great innovations /technology to help monitor our babies while they are sleeping and alert us of dangers.

Thank you in advance for your support. I would also like to ask my colleagues from med school, clinical rotations, residency and current co-workers to please reach out to me if you want to help with research or any contribution.

In the name of our beautiful Nash, lets Mash Out SIDS!



Source: Before it's News, April 4th 2016.



VAN UK's Comment: Dr Perry was told to stop fundraising for vaccine research and to take down his social media pages about it.

http://beforeitsnews.com/terrorism/2016/04/doctors-son-dies-of-sids-after-vaccines-2458119.html

http://vaccinecommonsense.com/2016/01/26/804/

Police Officer for 22 Years, Thrown Out of the Force for Whistleblowing about Vaccines and SIDS

I am an ex-Sgt of Police after 20 years in Queensland. Every SIDS mother told me their babies were healthy prior to vaccines and then deteriorated and died after.

Christopher William Savage

POLICE SERGEANT CHRIS SAVAGE BLOWS LID ON VACCINE CRIMES IN AUSTRALIA!!

[vid] POLICE SERGEANT CHRIS SAVAGE BLOWS LID ON VACCINE CRIMES IN AUSTRALIA!!

Mother Explains How Her Daughter Almost Became a SIDS Case when She Stopped Breathing after DTaP and Pneumonia Vaccines - Mother Did CPR to Revive Her

A modified self-controlled case series method to examine association between multidose vaccinations and death.

Abstract

The self-controlled case series method (SCCS) was developed to analyze the association between a time-varying exposure and an outcome event. We consider penta- or hexavalent vaccination as the exposure and unexplained sudden unexpected death (uSUD) as the event. The special situation of multiple exposures and a terminal event requires adaptation of the standard SCCS method. This paper proposes a new adaptation, in which observation periods are truncated according to the vaccination schedule. The new method exploits known minimum spacings between successive vaccine doses. Its advantage is that it is very much simpler to apply than the method for censored, perturbed or curtailed post-event exposures recently introduced. This paper presents a comparison of these two SCCS methods by simulation studies and an application to a real data set. In the simulation studies, the age distribution and the assumed vaccination schedule were based on real data. Only small differences between the two SCCS methods were observed, although 50 per cent of cases could not be included in the analysis with the SCCS method with truncated observation periods. By means of a study including 300 uSUD, a 16-fold risk increase after the 4th dose could be detected with a power of at least 90 per cent. A general 2-fold risk increase after vaccination could be detected with a power of 80 per cent. Reanalysis of data from cases of the German case-control study on sudden infant death (GeSID) resulted in slightly higher point estimates using the SCCS methods than the odds ratio obtained by the case-control analysis.



Source: Stat Med. 2011 Mar 15;30(6):666-77. doi: 10.1002/sim.4120. Epub 2010 Nov 30.

https://www.ncbi.nlm.nih.gov/pubmed/21337361

Dr Archie Kalokerinos Explains the Sudden Infant Death Syndrome and its Relation to Vaccination

Discussion starts several minutes (10) in as this is part of a much bigger interview, taken in 2004.

Vaccines and SIDS accounts on Camera